Content Archives. Joke SCPs - Some of the best articles on the site are clever and funny. Make sure yours is before you add it. Archived SCPs - SCPs preserved due to. Reader Results - Perfect Health Diet. I started feeling terrible in the winter of 2. I went to five doctors. I had blood drawn about 1. I was afraid I would be bedridden one day because of. The first doctor who helped me did so by diagnosing me with fibromyalgia. I was informed that my previous doctors did not believe fibromyalgia existed,since it could not be tested with a blood sample. A stroke is when poor blood flow to the brain results in cell death. There are two main types of stroke: ischemic, due to lack of blood flow, and hemorrhagic, due to. Any thing I need to watch for or consider? I know this is very natural but. The English version offers selected articles from. Original Article. A Low-Carbohydrate as Compared with a Low-Fat Diet in Severe Obesity. Frederick F. Samaha, M.D., Nayyar Iqbal, M.D., Prakash Seshadri, M.D., Kathryn. I began to have a disdainfor most doctors. He started me on Savella 5. I improved on Savella, and was told todo a cleanse diet to detect food sensitivities. Many people with fibromyalgiahave food sensitivities as well. I did not have the will power to executethis plan properly. However, I was not going to rely on doctors anymore, so Ibegan my search for healing on my own. I tried a few different things, natural supplements, and diets. Some gaveno help, some gave a bit of help, then I plateaued. Most importantly, withregard to the food restrictions I tried, I had such cravings that I did notcare about my fibromyalgia pain and just indulged. Then, when I was full, Icared about my fibromyalgia pain again. I learned of the Perfect Health Diet from a friend in the summer of 2. I did not think I would be able to do it all, knowing my failures in the past. So, I started small. I had a friend with celiac and was most convinced by thesection on the toxicity of wheat and other cereal grains. Thus, I began byeliminating these. I noticed an improvement within a few weeks: I could type! No more Dragon Naturally. Speaking for me! It was not carpal tunnel, in fact, Istill have bumps that come and go in my elbows and have to limit myweightlifting when I use my biceps in any way. ![]() Still trying to figure thatone out, but typing is not a problem anymore, as you can see here. In light of my previous failures, the key that made the avoidance of wheatand company successful was that I could eat a lot of fat. After a few weeks,I had some cravings for bread, but nothing as strong as my cravings forthings excluded in other trial diets. Now I have no cravings for bread. I thought all would be cured by avoiding gluten, so I started cutting backmy Savella dosage. When I got down to none, I felt fine for a week, then Islowly started to feel bad again. I got back on Savella, at 1/4 the originaldose. I decided I would put more effort into the other parts of the PHD. Next inline was vegetable oil. I had been eating a lot of salads, and I loved ranchdressing. With a few false starts, I finally broke the habit of vegetable oils,and was encouraged by some weight loss. Next, I went half in on the supplements. I noticed the magnesium hadsignificant effects on my muscle soreness and neck stiffness. Search settings; Web History : Advanced search Language tools. ![]() General Home Page Add a web site Jeff Bull Swap Meet Send a News Item Site Search by Hauser Racing 2017 FIA/FIM Championships schedule News, Coverage, Features. This page documents health changes our readers have experienced after adopting the Perfect Health Diet. If you have improved your health on our diet, please leave. The major risk of tight glucose control is hypoglycemia. Even among the relatively young and healthy population in the UKPDS trials, the incidence of hypoglycemia. Via Bleacher Report; Andrew Carter @Andrew. Jason Vargas now has 12 wins on the year. Huge outing for him and the team because of the double header tomorrow. I read the poston constipation and decided to add selenium, vitamin C, NAC, copper, etc. Finally, I got started on working up to high dose iodine, with therecommendation of starting low and doubling every month. In the last twoweeks I have been at 1. I have recently noticed a greatuptick in my mood and lowered occurrence of stiffness in my neck. I have beengetting much more done at work, and have not had as much “brainfog”. In fact, it took some forced thinking to recall how far I’ve come. I usedto stand up very slowly, and limp for a bit afterward because of pain in myhips. I used to try to play volleyball, but could not move suddenly or landthe wrong way lest I feel great pain. I used to accept that I would always bestiff and have difficulty moving in the morning. I used to get random painsin the bottom of my foot which made me limp. I used to take a lot of fiberand was still not regular. I used to be 2. 5 pounds heavier. I used to havestrong cravings for sugar, bread, chicken strips, and chips, just to name afew. I used to have acne flare ups all the time. In the past I had to stoptyping every five minutes and massage my elbows for ten minutes; it usuallyhurt to turn my neck; my shoulders were frequently sore; I could not throw abaseball, a football, or a frisbee. Frequently, I could not remember detailsof things I knew a lot about. I would get confused and get tired easily. Now, I do still have elbow pain and some psoriasis/rash, so all is notperfect. I am beginning to think these are unrelated to the fibromyalgia andthat the fibromyalgia is gone. I am at 1/3. 2nd of the original Savella dose,feeling fine, but will report back if I regress when taking the dose down tozero. THREE AND A HALF MONTHS LATER: I posted earlier about my progress with being on Savella for afibromyalgia diagnosis I received a bit over 2 years ago. With following the. PHD about 4. 0% or so I began to cut back on my medication. I did it too quickthe first time, and began to feel terrible. So, I stepped up the PHDcompliance of my diet as much as I could. I then began slowly cutting thedosage until I was off completely 3 months ago. I delayed reporting because I wanted to be sure symptoms would not flareup again. Today I ran my fastest 2 mile time, then played ultimate frisbeefor a few hours later in the day. I feel great. I used to get extremely sorefrom just trying to run half a mile, which was also pretty depressing. I amfeeling strong and limber with no trace of soreness. Stroke - Wikipedia. Stroke. Synonyms. Cerebrovascular accident (CVA), cerebrovascular insult (CVI), brain attack. CT scan of the brain showing a right- hemisphericischemic stroke. Specialty. Neurology. Symptoms. Inability to move or feel on one side of the body, problems understanding or speaking, feeling like the world is spinning, loss of vision to one side. There are two main types of stroke: ischemic, due to lack of blood flow, and hemorrhagic, due to bleeding. They result in part of the brain not functioning properly. If symptoms last less than one or two hours it is known as a transient ischemic attack (TIA) or mini- stroke. Other tests such as an electrocardiogram (ECG) and blood tests are done to determine risk factors and rule out other possible causes. Low blood sugar may cause similar symptoms. Aspirin should be used. Some hemorrhagic strokes benefit from surgery. Treatment to try to recover lost function is called stroke rehabilitation and ideally takes place in a stroke unit; however, these are not available in much of the world. About 8. 7% of strokes are ischemic, the rest being hemorrhagic. Bleeding can develop inside areas of ischemia, a condition known as . This definition was supposed to reflect the reversibility of tissue damage and was devised for the purpose, with the time frame of 2. The 2. 4- hour limit divides stroke from transient ischemic attack, which is a related syndrome of stroke symptoms that resolve completely within 2. There are four reasons why this might happen: Thrombosis (obstruction of a blood vessel by a blood clot forming locally)Embolism (obstruction due to an embolus from elsewhere in the body, see below). The Oxford Community Stroke Project classification (OCSP, also known as the Bamford or Oxford classification) relies primarily on the initial symptoms; based on the extent of the symptoms, the stroke episode is classified as total anterior circulation infarct (TACI), partial anterior circulation infarct (PACI), lacunar infarct (LACI) or posterior circulation infarct (POCI). These four entities predict the extent of the stroke, the area of the brain that is affected, the underlying cause, and the prognosis. The symptoms depend on the area of the brain affected. The more extensive the area of the brain affected, the more functions that are likely to be lost. Some forms of stroke can cause additional symptoms. For example, in intracranial hemorrhage, the affected area may compress other structures. Most forms of stroke are not associated with a headache, apart from subarachnoid hemorrhage and cerebral venous thrombosis and occasionally intracerebral hemorrhage. Different findings are able to predict the presence or absence of stroke to different degrees. Sudden- onset face weakness, arm drift (i. Similarly, when all three of these are absent, the likelihood of stroke is significantly decreased (– likelihood ratio of 0. A scoring system called ROSIER (recognition of stroke in the emergency room) is recommended for this purpose; it is based on features from the medical history and physical examination. Depending on the part of the brain affected, the defect in the brain is usually on the opposite side of the body. However, since these pathways also travel in the spinal cord and any lesion there can also produce these symptoms, the presence of any one of these symptoms does not necessarily indicate a stroke. In addition to the above CNS pathways, the brainstem gives rise to most of the twelve cranial nerves. A brainstem stroke affecting the brainstem and brain, therefore, can produce symptoms relating to deficits in these cranial nerves. Since blockage of the artery is gradual, onset of symptomatic thrombotic strokes is slower than that of a hemorrhagic stroke. A thrombus itself (even if it does not completely block the blood vessel) can lead to an embolic stroke (see below) if the thrombus breaks off and travels in the bloodstream, at which point it is called an embolus. Two types of thrombosis can cause stroke: Large vessel disease involves the common and internal carotid arteries, the vertebral artery, and the Circle of Willis. A stroke is the second leading cause of death in people under 2. An embolus is most frequently a thrombus, but it can also be a number of other substances including fat (e. Thus, the source of the embolus must be identified. Because the embolic blockage is sudden in onset, symptoms usually are maximal at the start. Also, symptoms may be transient as the embolus is partially resorbed and moves to a different location or dissipates altogether. Emboli most commonly arise from the heart (especially in atrial fibrillation) but may originate from elsewhere in the arterial tree. In paradoxical embolism, a deep vein thrombosis embolizes through an atrial or ventricular septal defect in the heart into the brain. The reduction could be to a particular part of the brain depending on the cause. It is most commonly due to heart failure from cardiac arrest or arrhythmias, or from reduced cardiac output as a result of myocardial infarction, pulmonary embolism, pericardial effusion, or bleeding. Because the reduction in blood flow is global, all parts of the brain may be affected, especially vulnerable . A watershed stroke refers to the condition when the blood supply to these areas is compromised. Blood flow to these areas does not necessarily stop, but instead it may lessen to the point where brain damage can occur. Venous thrombosis. Cerebral venous sinus thrombosis leads to stroke due to locally increased venous pressure, which exceeds the pressure generated by the arteries. Infarcts are more likely to undergo hemorrhagic transformation (leaking of blood into the damaged area) than other types of ischemic stroke. The hematoma enlarges until pressure from surrounding tissue limits its growth, or until it decompresses by emptying into the ventricular system, CSF or the pial surface. A third of intracerebral bleed is into the brain's ventricles. ICH has a mortality rate of 4. This may occur due to cocaine. Despite not causing identifiable symptoms, a silent stroke still damages the brain, and places the patient at increased risk for both transient ischemic attack and major stroke in the future. Conversely, those who have had a major stroke are also at risk of having silent strokes. Approximately 7. 70,0. MRI infarcts or hemorrhages. Silent strokes typically cause lesions which are detected via the use of neuroimaging such as MRI. Silent strokes are estimated to occur at five times the rate of symptomatic strokes. Since blood vessels in the brain are now blocked, the brain becomes low in energy, and thus it resorts into using anaerobic metabolism within the region of brain tissue affected by ischemia. Anaerobic metabolism produces less adenosine triphosphate (ATP) but releases a by- product called lactic acid. Lactic acid is an irritant which could potentially destroy cells since it is an acid and disrupts the normal acid- base balance in the brain. The ischemia area is referred to as the . This sets off a series of interrelated events that result in cellular injury and death. A major cause of neuronal injury is the release of the excitatory neurotransmitter glutamate. The concentration of glutamate outside the cells of the nervous system is normally kept low by so- called uptake carriers, which are powered by the concentration gradients of ions (mainly Na+) across the cell membrane. However, stroke cuts off the supply of oxygen and glucose which powers the ion pumps maintaining these gradients. As a result, the transmembrane ion gradients run down, and glutamate transporters reverse their direction, releasing glutamate into the extracellular space. Glutamate acts on receptors in nerve cells (especially NMDA receptors), producing an influx of calcium which activates enzymes that digest the cells' proteins, lipids, and nuclear material. Calcium influx can also lead to the failure of mitochondria, which can lead further toward energy depletion and may trigger cell death due to programmed cell death. These react with and damage a number of cellular and extracellular elements. Damage to the blood vessel lining or endothelium is particularly important. In fact, many antioxidant neuroprotectants such as uric acid and NXY- 0. Free radicals also directly initiate elements of the programmed cell death cascade by means of redox signaling. However, brain tissue is especially vulnerable to ischemia since it has a little respiratory reserve and is completely dependent on aerobic metabolism, unlike most other organs. In addition to damaging effects on brain cells, ischemia and infarction can result in loss of structural integrity of brain tissue and blood vessels, partly through the release of matrix metalloproteases, which are zinc- and calcium- dependent enzymes that break down collagen, hyaluronic acid, and other elements of connective tissue. Other proteases also contribute to this process. The loss of vascular structural integrity results in a breakdown of the protective blood brain barrier that contributes to cerebral edema, which can cause secondary progression of the brain injury. Some causes of hemorrhagic stroke are hypertensive hemorrhage, ruptured aneurysm, ruptured AV fistula, transformation of prior ischemic infarction, and drug induced bleeding. In addition, the pressure may lead to a loss of blood supply to affected tissue with resulting infarction, and the blood released by brain hemorrhage appears to have direct toxic effects on brain tissue and vasculature. Right image after 7 hours. Stroke is diagnosed through several techniques: a neurological examination (such as the NIHSS), CT scans (most often without contrast enhancements) or MRI scans, Doppler ultrasound, and arteriography. The diagnosis of stroke itself is clinical, with assistance from the imaging techniques. Imaging techniques also assist in determining the subtypes and cause of stroke.
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